Figure 1From: Downregulation of TES by hypermethylation in glioblastoma reduces cell apoptosis and predicts poor clinical outcomeTES promoter methylation status in glioblastoma (GBM, n = 42) and normal (n = 8) samples analyzed by DNA promoter methylation microarray. (A) Comparison of TES promoter methylation status in GBM and normal samples by microarray analysis (up) and independent t-test analysis (down). (B) Western blot of TES in U251 cell line before and after 5-aza-2-deoxycytidine treatment. Anti-tublin was used as a protein loading control.Back to article page