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Table 2 Comparison of the demographic and clinical data of patients in each group

From: Dysregulation of miR-210 is involved in the development of diabetic retinopathy and serves a regulatory role in retinal vascular endothelial cell proliferation

GroupAge (year)Gender (male/female)BMI (kg/m2)Disease course (year)FPG (mmol/l)TG (mmol/l)TC (mmol/l)HbA1c (%)FINS (mIU/I)HOMA-IR
HC50.85±5.2028/3221.81±2.205.14 ± 0.831.47±0.174.28±0.494.92±0.465.38±0.541.29±0.08
NDR50.23±3.8123/1721.78±1.844.27±0.726.15±0.58*1.50±0.144.40±0.516.61±0.59*5.41±0.651.57±0.10*
NPDR50.73±3.8028/3222.07±2.739.55±1.57#7.42±0.31*,#1.52±0.284.50±0.757.31±0.28*,#5.46±0.791.61±0.28*
PDR51.40±3.9326/2422.61±3.1212.68±1.43#,&8.51±0.44*,#,&1.55±0.264.57±0.738.00±0.34*,#,&5.51±0.631.84±0.23*,#,&
  1. Data are expressed as the mean ± SD or number. Multiple comparisons were made using ANOVA followed by Tamhane’s T2 tests, while the gender distribution among groups were evaluated by using Chi-square test
  2. HC healthy control, NDR DM without DR patients, NPDR non-proliferative DR, PDR proliferative DR, FPG fasting plasma glucose, TG triacylglycerol, TC total cholesterol, HbA1c glycosylated hemoglobin, FINS fasting insulin, HOMA-IR homoeostasis model assessment of insulin resistance
  3. *P < 0.05 compared with HC group; #P < 0.05 compared with NDR group; &P < 0.05 compared with NPDR group