Skip to main content
Fig. 7 | European Journal of Medical Research

Fig. 7

From: AMP-activated protein kinase α2 contributes to acute and chronic hyperuricemic nephropathy via renal urate deposition in a mouse model

Fig. 7

Schematic representation of AMPK α2 contributing to acute and chronic hyperuricemic nephropathy. AMPK α2 suppresses uric acid excretion through uric acid transporters. Accumulation of uric acid increased uric acid deposition, which further leads to the formation of a renal small tube type, inflammatory cell infiltration, and renal fibrosis

Back to article page

European Journal of Medical Research

ISSN: 2047-783X

Contact us