Fig. 2
miR-217 inhibits the proliferation of endometrial cancers through by miR-217/MARK1 axis. Ishikawa cell was transfected with control mimic-NC and miR-217 mimic for 48Â h and subjected to RT-qPCR analysis to examine the presence of miR-217 (A). Cell proliferation was evaluated by CCK-8 assay (B) and colony formation assay with statistics (C, D) after transfection. The migration and invasion capacity of Ishikawa cell cells was determined by Transwell migration and invasion assay after transfection (E). The statistics of Transwell migration (F) and invasion (G) assay were analyzed. Western blot was applied to examine the level of MARK1 protein with an anti-MARK1 antibody (H) after transfection with mimic-NC and miR-217 mimic. The statistics of WB were analyzed (I)