Table 2 List of models for myocardial stress tolerance modulated by O-GlcNAc modification

In vitro

Heat shock

NRVMs

Increase

Increase

N/A

[63]

Hypoxia and H/R

NRCMs, NMCMs, CSCs

Increase

Increase

P, G

[46, 50, 51, 64]

CoCl₂

HUVECs

N/A

N/A

G

[65]

I/R

NRVMs

Increase

Increase

M, P, G

[61, 66,67,68,69]

H₂O₂

NRCMs

Complex

Increase

P, G

[51, 66, 70]

ER stress (BfA, TM)

NRCMs

Increase

Increase

P, G

[71]

LPS

NRVMs, macrophages

N/A

N/A

M, P, G

[23]

TNF-α

Aortic rings, HUVECs

VSMCs

N/A

Increase

M, P

[72, 73]

Ex vivo

I/R, IPC, rIPC

Isolated hearts

Increase

Increase

M, P

[9, 34, 42, 62, 74,75,76]

In vivo

I/R, IPC

Mice, Murine

Complex

Increase

P

[51, 68,69,70, 77]

Hypoxia/ hypoxic acclimation

Mice

Increase

Increase

P

[65, 77]

Acute arterial injury

Rats

Complex

Increase

M, P

[72, 78]

Trauma- haemorrhage

Rats

Decrease

Increase

M, P, G

[23, 36, 37, 79]

Septic shock

Rats

N/A

Increase

P

[38]